Respirable Crystalline Silica (RCS) has been used for centuries in many construction materials including sand, stone, concrete, and brick. The benefits of RCS lie in its strength, durability, and abundance, making it indispensable in activities like masonry, sandblasting, and cutting or grinding engineered and natural stone. With the rise of engineered stone in the late 20th century, especially in countertops and flooring, the use of RCS became even more prominent due to the material’s workability and aesthetic appeal.
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26.11.2024
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Concerns about the health risks of silica dust exposure began to gain traction in the early 20th century, with links to lung diseases like silicosis becoming more apparent as industrial processes involving RCS increased. However, it wasn’t until the 1930s that significant attention was drawn to silicosis during the construction of the Hawk’s Nest Tunnel in the United States, which resulted in one of the worst industrial health disasters, with hundreds of workers affected.
In recent decades, the risks of RCS exposure have become better understood globally. Engineered stone, in particular, has raised alarms due to its high silica content (often exceeding 90%), and the process of dry cutting which releases hazardous respirable silica dust into the air. Serious concerns about silicosis re-emerged in the 2010s as cases spiked in countries such as Australia, Spain, and Israel. This has prompted stronger regulation and, in some jurisdictions, even bans on the use of engineered stone to mitigate the risks associated with working with RCS.
RCS and Silicosis
Silica dust causes silicosis through a process that begins when tiny respirable crystalline silica (RCS) particles are inhaled into the lungs. These particles are released into the air during activities like cutting, grinding, or drilling materials such as concrete, stone, or engineered stone. Once inhaled, the fine particles bypass the body’s natural defences in the upper respiratory tract and settle deep within the lungs in the alveoli, the tiny air sacs where oxygen exchange occurs.
The Mechanism of Damage
The sharp, crystalline particles then irritate and damage lung tissue, and the immune system responds by sending macrophages (a type of white blood cell) to engulf and remove the particles. The macrophages often fail to break down the silica particles and may themselves die, releasing inflammatory substances. This triggers the production of fibrous scar tissue (fibrosis) in the lungs, which thickens and stiffens the alveoli, reducing their elasticity.